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presented a heterozygous CHAC1 deletion mouse model that was protected from kidney disease progression across three different disease models, without any known phenotypic changes under control conditions ( Despite its injury-promoting effects in some organs, CHAC1 contributes to tissue repair in certain contexts

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Note that the Kaplan-Meier survival curves were generated using samples whose expression levels were among the top and bottom 33% of expression values for corresponding genes

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